August192014
11AM

(Source: best-of-memes)

August182014
psych2go:

For more posts like these, go visit psych2go
Psych2go features various psychological findings and myths. In the future, psych2go attempts to include sources to posts for the purpose of generating discussions and commentaries. This will give readers a chance to critically examine psychology.
Fact submitted by: bonjourtammy

psych2go:

For more posts like these, go visit psych2go

Psych2go features various psychological findings and myths. In the future, psych2go attempts to include sources to posts for the purpose of generating discussions and commentaries. This will give readers a chance to critically examine psychology.

Fact submitted by: bonjourtammy

(via psych-facts)

6PM
ultrafacts:

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ultrafacts:

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6PM
ucsdhealthsciences:

Aspirin, Take TwoWhite blood cell research shows how causing and conquering inflammation are inextricably linked
Hugely popular non-steroidal anti-inflammation drugs like aspirin, naproxen (marketed as Aleve) and ibuprofen (Advil, Motrin) all work by inhibiting or killing an enzyme called cyclooxygenase – a key catalyst in production of hormone-like lipid compounds called prostaglandins that are linked to a variety of ailments, from headaches and arthritis to menstrual cramps and wound sepsis.
In a new paper, published this week in the online early edition of PNAS, researchers at the University of California, San Diego School of Medicine conclude that aspirin has a second effect: Not only does it kill cyclooxygenase, thus preventing production of the prostaglandins that cause inflammation and pain, it also prompts the enzyme to generate another compound that hastens the end of inflammation, returning the affected cells to homeostatic health.
“Aspirin causes the cyclooxygenase to make a small amount of a related product called 15-HETE,” said senior author Edward A. Dennis, PhD, Distinguished Professor of Pharmacology, Chemistry and Biochemistry. “During infection and inflammation, the 15-HETE can be converted by a second enzyme into lipoxin, which is known to help reverse inflammation and cause its resolution – a good thing.”
Specifically, Dennis and colleagues looked at the function of a type of white blood cells called macrophages, a major player in the body’s immune response to injury and infection. They found that macrophages contain the biochemical tools to not just initiate inflammation, a natural part of the immune response, but also to promote recovery from inflammation by releasing 15-HETE and converting it into lipoxin as the inflammation progresses.
Dennis said the findings may open new possibilities for anti-inflammatory therapies by developing new drugs based on analogues of lipoxin and other related molecules that promote resolution of inflammation. “If we can find ways to promote more resolution of inflammation, we can promote health,” he said.  
Image: Scanning electron micrograph of macrophage. Image courtesy of National Cancer Institute.

ucsdhealthsciences:

Aspirin, Take Two
White blood cell research shows how causing and conquering inflammation are inextricably linked

Hugely popular non-steroidal anti-inflammation drugs like aspirin, naproxen (marketed as Aleve) and ibuprofen (Advil, Motrin) all work by inhibiting or killing an enzyme called cyclooxygenase – a key catalyst in production of hormone-like lipid compounds called prostaglandins that are linked to a variety of ailments, from headaches and arthritis to menstrual cramps and wound sepsis.

In a new paper, published this week in the online early edition of PNAS, researchers at the University of California, San Diego School of Medicine conclude that aspirin has a second effect: Not only does it kill cyclooxygenase, thus preventing production of the prostaglandins that cause inflammation and pain, it also prompts the enzyme to generate another compound that hastens the end of inflammation, returning the affected cells to homeostatic health.

“Aspirin causes the cyclooxygenase to make a small amount of a related product called 15-HETE,” said senior author Edward A. Dennis, PhD, Distinguished Professor of Pharmacology, Chemistry and Biochemistry. “During infection and inflammation, the 15-HETE can be converted by a second enzyme into lipoxin, which is known to help reverse inflammation and cause its resolution – a good thing.”

Specifically, Dennis and colleagues looked at the function of a type of white blood cells called macrophages, a major player in the body’s immune response to injury and infection. They found that macrophages contain the biochemical tools to not just initiate inflammation, a natural part of the immune response, but also to promote recovery from inflammation by releasing 15-HETE and converting it into lipoxin as the inflammation progresses.

Dennis said the findings may open new possibilities for anti-inflammatory therapies by developing new drugs based on analogues of lipoxin and other related molecules that promote resolution of inflammation. “If we can find ways to promote more resolution of inflammation, we can promote health,” he said.  

Image: Scanning electron micrograph of macrophage. Image courtesy of National Cancer Institute.

6PM

(Source: best-of-memes)

6PM
6PM
ultrafacts:

01paige01:

ultrafacts:

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Cats should get more credit for being cats.

The WHOLE internet is dedicated to cats already

ultrafacts:

01paige01:

ultrafacts:

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Cats should get more credit for being cats.

The WHOLE internet is dedicated to cats already

4PM
“You don’t always win your battles, but it’s good to know you fought.” Lauren Bacall (via glamour)
4PM

(Source: best-of-memes)

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